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Histologically, depending on the extent of tubular gland formation versus fingerlike villous projections, they are classified as tubular adenomas, villous adenomas, or tubulovillous adenomas. 42 CASE FILES: PATHOLOGY Figure 4-1. Colonic polyp: gross pictograph. Polyposis and Inherited Colon Cancer Syndromes Syndromes that involve the formation of multiple gastrointestinal polyps occur infrequently. Some, such as Peutz-Jeghers syndrome and Cowden disease, are autosomal dominant, resulting in the formation of nonneoplastic hamartomatous polyps; others, such as Canada-Cronkhite syndrome, are not hereditary and result in multiple juvenile polyps.

If anti-hepatitis C antibody is present, acute hepatitis C is diagnosed, but the test may be negative for several months. The hepatitis C polymerase chain reaction (PCR) assay, which becomes positive earlier in the disease course, often aids in the diagnosis. Acute hepatitis B infection is diagnosed by the presence of hepatitis B surface antigen (HBsAg) in the clinical context of elevated serum transaminase levels and jaundice. HBsAg later disappears when the antibody (anti-HBs) is produced (see Figure 3-2).

The hepatitis C polymerase chain reaction (PCR) assay, which becomes positive earlier in the disease course, often aids in the diagnosis. Acute hepatitis B infection is diagnosed by the presence of hepatitis B surface antigen (HBsAg) in the clinical context of elevated serum transaminase levels and jaundice. HBsAg later disappears when the antibody (anti-HBs) is produced (see Figure 3-2). There is often an interval of a few weeks between the disappearance of HBsAg and the appearance of anti-HBsAb, which is referred to as the window period.

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